Appendix II, classification of symptoms of poisoning plant

.RG Pharmaceutica though that plants containing cyanide precursor of (cyanogenic plants): nucleolus as cassava, almonds, flax, cycads, peach, cherry, plum, plum, loquat fruit, the particularly raw goods. As bitter almonds contain amygdalin (amygdalin) 3%, children eating 10-180 grains can cause poisoning; the general market

Appendix

of sweet almonds contains amygdalin about 0.11%. Almonds, walnuts, ginkgo and other toxic cyanide is produced by strongly with Fe3 + (ferric) combine to prevent the reduction of cytochrome oxidase (cytochrome oxidase) of the electron transport system related to ferrous (Fe2 +, ferrous) ion type . Electron transfer from cytochrome to oxygen molecules is hindered, and then ATP production is suppressed, forcing the cells to produce energy by anaerobic metabolism. As in all cases of hypoxia, anaerobic glycolysis (glycolysis) can only produce a small amount of ATP and a lot of lactic acid. Thus cyanide poisoning victims show signs and symptoms of hypoxia. Similar to carbon monoxide poisoning, cyanide toxicity primarily affects the height of oxygen organ. However, the starting time of such signs and symptoms, depending on the route of exposure vary, but usually faster. Inhalation can cause rapid death, but in feeding mode, it will delay symptoms for 30 minutes or more. CNS dysfunction can cause loss of consciousness and respiratory arrest. Lab found that its performance for hypoxia and typically include severe anion gap acidosis produced by the lactic acid